Devido a questões de autoria, a ECS News apenas pode disponibilizar o abstract do dito artigo:
Targaryen H, Cricas L, Tank W, Killington B, Monster69 C, Killer S, Borrado E
The authors declare that their conflits of interest are solved through Tank Trouble, with no additional legal action required.
Abstract:
The renis (singular - reni) are paired organs in the brain of most mammals. They are located below the supraorbital notch, anteriorly to the frontal cerebral cortex. Their function is to filtrate the cerebrospinal fluid, earning them the analogy of being the kidneys of the brain.
The renis are the human organ with the highest concentration of noobocytes. Administration of noobin into animal models shows that the protein is captured by the NO-OB37 receptors present in the plasmatic membrane of the noobocytes. These receptors are putatively stimulated by gymin, a protein coded by the gene FAG69 in the short arm of chromosome 13. In several studies, animals treated with gymin showed overexpressed numbers and activity of NO-OB. Conversely, animals taking videogamin showed an abrupt noobocyte decrease.
In this study, we aim to evaluate the capacity of sousocytes, the most well-known type of noobocytes, when it comes to implementing noobin. High noobin capture is thought to be linked with an increased secretion of failin, another inhibitory target for the Videogamin-Noexercisin-Juschillin Axis.
Sample sousocytes from rat renis were kept in a favorable environment, conserved at frost temperature. They were then treated with different combinations of noobin, gymin and videogamin. Noexercisin and juschillin, functionally similar proteins to videogamin, were not used due to their less effective clinical use - both were vastly outclassed by videogamin when it came to preventing noobism, a severe mental disease affecting motor prowess and decision-making.
Results suggest that noobin works as a tetrameric enzyme complex, catalyzing the formation of failin. Noobin concentration practically did not fall off, indicating that it has an extremely durable turnover and can keep catalyzing almost indefinitely. Gymin by itself achieved similar results, albeit not as spectacular. When the two were combined, failin concentration was higher than ever, suggesting some cooperativity between the two proteins. Currently, the most accepted theory is that gymin inhibits videogamin, therefore also inhibiting its inhibitory action against noobin. The models treated with videogamin showed an expected decrease in failin concentration.
Our results are consistent with previous studies, and further confirm that videogamin is indeed the best way to deal with short-term noobism. In long-term noobism, it is assumed that the neurons have sustained so much damage that they are oversensitive to failin.
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